Titlebook: Cardiovascular Disease, Volume 1; Genetics Qing K. Wang Book 2007 Humana Press 2007 Chromosom.bioinformatics.cardiovascular.gene therapy.ge

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Genome-Wide Association Study to Identify Single-Nucleotide Polymorphisms Conferring Risk of Myocarght result from the interactions of multiple genetic and environmental factors, none of which can cause disease solely by themselves. To reveal the genetic bases of MI, we performed a large-scale, case-control association study using 92,788 gene-based single-nucleotide polymorphism (SNP) markers. We

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Mutation Detection in Congenital Long QT Syndrome,arrhythmogenic disorders such as long QT syndrome (LQTS), catecholaminergic polymorphic ventricular tachycardia (CPVT), Andersen-Tawil syndrome, Brugada Syndrome, and Timothy syndrome. In addition, the genetic basis for cardiomyopathic processes vulnerable to sudden arrhythmic death—hypertrophic car

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https://doi.org/10.1007/978-3-642-24226-7ns, and other complicated chromosomal changes. Fluorescence . hybridization (FISH), a technique involving hybridization of labeled probes to chromosomes and detection of hybridization via fluorochromes, has become a popular method for identification and characterization of cytogenetic abnormalities.

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https://doi.org/10.1007/978-3-642-24226-7complex clinical phenotypes. Comparative genomic hybridization by microarray (array CGH) is a high-throughput and high-resolution method for the detection of microscopic and submicroscopic chromosome abnormalities, some of which may not be detectable by conventional cytogenetic techniques. In additi

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