Titlebook: Cardiovascular Complications in Cancer Therapy; Antonio Russo,Giuseppina Novo,Fausto J. Pinto Book 2019 Springer Nature Switzerland AG 201

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Ordnance

acrophobia

Heart Failure and Left Ventricular Dysfunction,cause is, are to improve their clinical status, functional capacity, and quality of life, prevent hospital admission, and reduce mortality. However, the real challenge of cardio-oncology is to avoid that damage occurs at all..For this reason early diagnosis of myocardial impairment is crucial, and i

conspicuous

Peripheral Artery Disease and Stroke,longer time of drug exposure, and sequential use of potent TKIs. In addition, circulating concentrations of VEGF are reduced by cyclophosphamide administered at continuous low doses, which might underpin some of the observed vascular toxicity, as seen in patients treated with VEGFIs. This alkylating

Certainty

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Atrial Fibrillation in Cancer,cally every form of cardiac or vascular disease. Arrhythmias represent a significant part of this spectrum, and atrial fibrillation (AF) is one of the main arrhythmias occurring in cancer patients..Several studies have documented the relationship between cancer and AF. In terms of epidemiology, AF i

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的事物

Diagnosis of Cardiac Damage: Role of Stress Echo,motherapy, a complete evaluation of systolic and diastolic function at rest and after stress should be performed. Contractile reserve assessed by either ejection fraction or speckle tracking echocardiography should be determined considering its diagnostic and prognostic power. New technologies appli

大量殺死

Early Detection and Monitoring of Vascular Damage, Accordingly, promptly detection of these possible adverse effects by vascular imaging should be strongly promoted to introduce efficient prophylactic therapies when indicated. It is fundamental to diagnose vascular complications and treat them aggressively in order to reduce deleterious adverse eff

penance

Prevention and Clinical Management of Cardiovascular Damage Induced by Anticancer Drugs: Need for Etion of a large number of molecular features and have the potential to identify new factors that contribute to cardiac and endothelial function and how they interact. These technologies could play a pivotal role in unraveling the pathophysiology of vascular damage induced by anticancer treatment, in