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Titlebook: Cardiorenal Syndrome in Heart Failure; W. H. Wilson Tang,Frederik H. Verbrugge,Wilfried M Book 2020 Springer Nature Switzerland AG 2020 Ca

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發(fā)表于 2025-3-21 19:08:56 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Cardiorenal Syndrome in Heart Failure
編輯W. H. Wilson Tang,Frederik H. Verbrugge,Wilfried M
視頻videohttp://file.papertrans.cn/222/221891/221891.mp4
概述Features a multidisciplinary approach with contributions from both cardiologists and nephrologists.Contains a practically applicable case-based approach to managing patients with refractory congestion
圖書封面Titlebook: Cardiorenal Syndrome in Heart Failure;  W. H. Wilson Tang,Frederik H. Verbrugge,Wilfried M Book 2020 Springer Nature Switzerland AG 2020 Ca
描述.This comprehensive manual reviews the management of cardiorenal syndrome in heart failure. Chapters are structured in a practically applicable and easy-to-follow format with realistic case vignettes and key clinical management questions and answers, followed by a brief discussion of underlying pathophysiological mechanisms of a patient with cardiorenal syndrome. Building from this case, key questions are posed that are relevant to the clinical management and then potential evidence-based treatment strategies are proposed. Topics covered include loop diuretic resistance in acute and chronic heart failure, abdominal congestion, low output failure and potential diuretic complications due to hyponatremia.?..Cardiorenal Syndrome in Heart Failure. thoroughly reviews cardiorenal syndrome from the perspective of both the cardiologist and nephrologist. Its case-based approach makes it an ideal resource for both practising and trainee cardiology and nephrology practitioners.?.
出版日期Book 2020
關鍵詞Cardio-Renal Syndrome; Loop diuretic resistance; Heart Failure; Diuretics; Ultrafiltration; chronic kidne
版次1
doihttps://doi.org/10.1007/978-3-030-21033-5
isbn_softcover978-3-030-21035-9
isbn_ebook978-3-030-21033-5
copyrightSpringer Nature Switzerland AG 2020
The information of publication is updating

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P. Bach,S. Cluzeau,C. Lambermontuts into central autonomic nuclei responsible for cardiovascular control and sympathetic outflow. In disease states such as heart failure and renal failure, increased renal sympathetic nerve activity can further worsen renal function, with a shift from inhibitory to “l(fā)ess inhibitory” (or potentially
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https://doi.org/10.1007/978-1-349-26729-3ir effectiveness or induce worrisome worsening of renal function. It is important to understand and leverage the pharmacokinetic and pharmacodynamic properties of diuretic drugs, if therapeutic success is to be achieved. At its simplest, a diuretic must be administered or ingested and absorbed, must
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https://doi.org/10.1007/978-1-349-26729-3ent hemodialysis itself causes significant hemodynamic stress and vascular changes as well as a potential high-output state from arteriovenous fistula that can be linked to left ventricular hypertrophy and myocardial ischemia and stunning. Cardiovascular and metabolic changes such as fluid overload,
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https://doi.org/10.1007/978-1-349-26729-3chieve symptomatic relief. The management of heart failure through the use of intravenous diuretics is at times limited due to renal function. An elevated Cr level, which has previously been associated with poor outcomes, is a common end-point in most AHF trials. However, recent data has called into
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