Titlebook: Cardio-Nephrology; Confluence of the He Janani Rangaswami,Edgar V. Lerma,Claudio Ronco Book 2017 Springer International Publishing AG 2017

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Erythropoietin and Its Cardiovascular Effectsave non-hematological effects on cardiac myocytes, angiogenesis, and endothelial cells. Several post-approval studies of erythropoietic stimulating agents (ESAs) examined patient outcomes related to target Hb level and most demonstrated increased adverse cardiovascular outcomes with a “normal” Hb ta

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Pathophysiology of the Cardiorenal Syndromes Types 1–5: Updates from the Eleventh Consensus Conferen, the term cardiorenal syndrome (CRS) has been used to define different clinical conditions in which heart and kidney dysfunction overlap. Type 1 CRS (acute cardiorenal syndrome) is characterized by acute worsening of cardiac function leading to AKI (Ronco in Contrib Nephrol 164:33–38, .; Eren et al

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thyroid-hormone

Kidney Attack: Is NGAL Set to Take the Stage with Troponins?se to man have consistently revealed NGAL to be one of the most dramatically upregulated genes in the kidney soon after an ischemic or nephrotoxic insult. The 25?kDa NGAL protein is also highly induced in regenerating and recovering kidney tubule cells, where it exerts a robust nephron-protective ef

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Ultrafiltration Therapy in Decompensated Heart Failure between fluid removal via UF and fluid removal by diuretics. Clinical trials comparing UF and diuretics in patients with decompensated heart failure are not uniform in their design or their results. Future investigations of this therapy should focus on specific subgroups of patients with HF and sho

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https://doi.org/10.1007/978-3-319-56042-7Anticoagulation and antiplatelet therapy in CKD; Cardiac evaluation of the renal transplant candidate

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Osteoarthritis

Menschliche Wahrnehmung: Die Sicht der Dingeic stimulus for endothelial nitric oxide (NO) synthesis is blood flow-induced shear stress, a process termed “flow-mediated vasodilatation”, which is a clinical assessment of endothelial well-being. Endothelial cell damage or injury is invariably associated with thrombosis, hypertension, and atheros

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Teamarbeit: Der Schlüssel zum Erfolg with CKD, vascular and valvular calcification result from an increase in calcium deposition due to metabolism confluence of inhibitory regulation of calcium deposition, increased matrix metalloproteinases (MMP) concentration, chronic inflammation, and the effect of mechanical stress. Vascular calci