Titlebook: Cardiac Ischemia: From Injury to Protection; Bohuslav O?t’ádal,Franti?ek Kolá? Book 1999 Springer Science+Business Media New York 1999 ada

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美色花錢

Siegfried Carl,Vy Khoi Le,Dumitru Motreanulterations of the capillary network in an independent or combined fashion create varying degrees of ischemic injury. Its extent depends on the severity and duration of flow deprivation; ischemia will initiate a sequence of progressively more severe events which, unless halted by reperfusion, will ev

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dermatomyositis

Nonsmooth Mathematical Programming Problems,ygen deprivation (Fig. 25). It is, therefore, not surprising that the interest of many experimental and clinical cardiologists during the past 35 years has been focused on the question of how cardiac tolerance to oxygen deprivation might be increased. The relatively short history of this endeavour r

CANE

Nonconvex Optimization and Its Applicationsnsequences may be essentially influenced by risk factors acting already during development. Accordingly, the experimental studies of the pathogenetic mechanisms of cardiac ischemia must shift to the early ontogenetic periods.

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https://doi.org/10.1007/978-1-4757-3025-8adaptation; apoptosis; calcium; cells; death; development; heart; hypoxia; metabolism; necrosis; prevention; re

惡名聲

978-1-4419-5105-2Springer Science+Business Media New York 1999

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MAOIS

Reperfusion-Induced Injury,In clinical therapy of evolving acute myocardial infarction, early coronary reperfusion has proved to be the only way to limit infarct size. However, there is also evidence from animal studies that reperfusion may contribute to further tissue damage, a phenomenon known as “reperfusion injury”.

Scintillations

Myocardial Hypoxia and Ischemia,bring about a substantial improvement in oxygen supply by the increased extraction of oxygen from the blood; thus the only way of meeting the higher oxygen demand is through an increased blood supply (Fig. 1). Theoretically, any of the known mechanisms leading to tissue hypoxia can be responsible fo