Titlebook: Cardiac Energy Metabolism in Health and Disease; Gary D. Lopaschuk,Naranjan S. Dhalla Book 2014 Springer Science+Business Media New York 2

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Nonparametric Functional Data Analysisnagement predisposes the patient to heart failure, which is the leading cause of diabetes related deaths. One instigator for this cardiac dysfunction is change in fuel utilization by the heart. Thus, following diabetes, when cardiac glucose utilization is impaired, the heart undergoes metabolic tran

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https://doi.org/10.1007/0-387-36620-2l carbohydrate (glucose, lactate) uptake have been unraveled in detail in the 1990s, insight into fatty acid uptake originates from more recent studies. Fatty acid movement across the sarcolemma is facilitated by membrane-associated proteins, specifically CD36, membrane-associated fatty acid-binding

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Small Ball Probabilities and Semi-metricsresult in dramatic alterations in cardiac energy metabolism, which can contribute to the progression of heart failure. Elevated rates of fatty acid oxidation and depressed rates of glucose oxidation characterize the cardiac metabolic profile in the setting of obesity-induced insulin resistance and d

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Local Weighting of Functional Variablesthat a complex network of nuclear transcription factors is essential for the coordinated regulation of mitochondrial biogenesis, maturation and function. These transcription factors guide developmental changes in mitochondrial number, structure, and dynamics as well as respond to various physiologic

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Nonparametric Functional Data Analysissm with efficient ATP generation and utilization. As ATP is primarily derived from mitochondrial oxidative phosphorylation, myocardial oxygen consumption (MVO.) can be used to measure the rate of energy expenditure of the heart. Although cardiac efficiency is an ambiguous term, it commonly embraces

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https://doi.org/10.1007/0-387-36620-2 function. Non-enzymatic and enzymatic mechanisms appear to function in facilitating this modification of protein lysine residues. The functional role of protein acetylation was originally linked to lifespan regulation in simple organisms under the control of sirtuin deacetylase enzymes. In higher o

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