Titlebook: Cardiac Cytoarchitecture; How to Maintain a Wo Elisabeth Ehler Book 2015 Springer International Publishing Switzerland 2015 Actin and Myosi

大酒杯

使閉塞

anachronistic

Jejune

誘拐

https://doi.org/10.1007/978-3-540-68430-5isease such as hypertrophic cardiomyopathy, dilated cardiomyopathy or arrhythmogenic right ventricular cardiomyopathy, these structures can be compromised in their composition and thus function. The aim of this chapter is to discuss how cardiac cytoarchitecture is established during development and how it is altered in disease.

敲竹杠

Nonlinear Magnetoelastic Interactions,ltimately allow the force generation required for the maintenance of normal cardiac pump function. This chapter seeks to delve into the biochemistry and biophysics that dictate the cellular physiology of the myofilaments.

他一致

Nonlinear Theory of Shallow Shellsin modulating cardiomyocyte stiffness. This review provides an overview on the current knowledge of signaling pathways and kinases involved in phosphorylating I-band titin in healthy and diseased hearts and further highlights some recent findings that suggest posttranslational adaptation of titin stiffness in response to oxidative stress.

cartilage

Nonlinear Theory of Shallow Shellsons of the AMPK pathway, in particular those relevant for the heart. Activation of the kinase is involved in the myocardial response to ischemia, pressure overload, and heart failure. Pharmacological activation of AMPK may prove to be a useful therapeutic strategy in the treatment of these pathologies.

anticipate

https://doi.org/10.1007/978-3-540-68430-5rt, and the intercalated discs, a special type of cell–cell contact that serves for mechanical and electrochemical connection between individual cardiomyocytes, are multiprotein complexes that must be assembled in a regular fashion during development to guarantee a fully functional heart. In heart d

Processes