Titlebook: Cardiac Arrhythmias: The Management of Atrial Fibrillation; Ronald W. F. Campbell,Michiel J. Janse Conference proceedings 1992 Springer-Ve

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別炫耀

Atrial Fibrillation: Is Our Electrophysiological Understanding on the Right Wavelength?,a is due to a focus that discharges so rapidly that uniform excitation of the atrium is no longer possible. This kind of fibrillation is best described as “fibrillatory conduction” where at multiple and varying sites rate-dependent arcs of conduction block develop (Allessie et al. 1985). To explain

indigenous

Class I Antiarrhythmic Drug Effects: What Is the Basis for Subgroups Ia, Ib and Ic in Human Heart Mtransmembrane potentials using conventional microelectrode techniques. Class I drugs, such as quinidine, share the property of depressing phase zero of the action potential by their ability to decrease the membrane conductance for Na.. Class II agents, such as propranolol, are characterized by their

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https://doi.org/10.1007/978-3-0348-8895-0a is due to a focus that discharges so rapidly that uniform excitation of the atrium is no longer possible. This kind of fibrillation is best described as “fibrillatory conduction” where at multiple and varying sites rate-dependent arcs of conduction block develop (Allessie et al. 1985). To explain

DUST

Pseudomonotone or weakly continuous mappingstransmembrane potentials using conventional microelectrode techniques. Class I drugs, such as quinidine, share the property of depressing phase zero of the action potential by their ability to decrease the membrane conductance for Na.. Class II agents, such as propranolol, are characterized by their

DUST

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