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Titlebook: Canine Parvovirus: A New Pathogen; Cherelyn Vella,S. W. Ketteridge Book 1991 Springer-Verlag Berlin Heidelberg 1991 AIDS.Antigen.Darminfek

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發(fā)表于 2025-3-21 20:00:11 | 只看該作者 |倒序瀏覽 |閱讀模式
書目名稱Canine Parvovirus: A New Pathogen
編輯Cherelyn Vella,S. W. Ketteridge
視頻videohttp://file.papertrans.cn/222/221305/221305.mp4
圖書封面Titlebook: Canine Parvovirus: A New Pathogen;  Cherelyn Vella,S. W. Ketteridge Book 1991 Springer-Verlag Berlin Heidelberg 1991 AIDS.Antigen.Darminfek
描述Canine parvovirus (CPV), a new virus of the Canidae firstappeared during the 1970s, but disease caused by this viruswas not reported until 1978.Then within a few months thevirus crossed national and continental boundaries causinghigh fatality in domestic dog populations. A similar patternof transmission was seen in sylvatic canines but infectionwaslargely subclinical. Between 1979 and 1981 CPV wasreplaced worldwide by an antigenic variant designatedCPV-2a. The epidemiological advantage of thisvariant overCPV is not understood.CPV is a new pathogen of dogs thathas attracted animpressive body of research. However aspects of thebiologyof this virus remain unclear, not least of which is thequestionof its possible origin. This book presents an up todate and comprehensivereview of the natural history of CPVand its control by prophylacticvaccination. Particularattention is paid to the techniques used to examine therelationship between CPV and other parvoviruses. CPV isthought tobe a variant of the virulent cat virus FPV(feline parvovirus) and at least one other variant is knownMEV (mink enteritis virus). Theories on thegenesis of CPVare discussed. An attempt to correlate the evidence anda
出版日期Book 1991
關鍵詞AIDS; Antigen; Darminfektion; Enteric Disease; Epidemiologie; Epidemiology; Myocarditis; Myokarderkrankung;
版次1
doihttps://doi.org/10.1007/978-3-642-76797-5
isbn_softcover978-3-540-54314-5
isbn_ebook978-3-642-76797-5
copyrightSpringer-Verlag Berlin Heidelberg 1991
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沙發(fā)
發(fā)表于 2025-3-21 21:27:41 | 只看該作者
Video and Opto-Digital Imaging Microscopy,ty that there may be some potential for CPV to spread into further species unrelated to the dog. At present, though, there is no irrefutable evidence of CPV causing disease in any other species of animal.
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Prospects and Problems,ty that there may be some potential for CPV to spread into further species unrelated to the dog. At present, though, there is no irrefutable evidence of CPV causing disease in any other species of animal.
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Book 1991ithin a few months thevirus crossed national and continental boundaries causinghigh fatality in domestic dog populations. A similar patternof transmission was seen in sylvatic canines but infectionwaslargely subclinical. Between 1979 and 1981 CPV wasreplaced worldwide by an antigenic variant designa
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New Horizons for Magic-Angle Spinning NMR,81, enteric disease caused by CPV had become world-wide, and a second manifestation, myocarditis, was recognised in young puppies only. At the time of its sudden appearance in 1978, canine populations simply had no immunity to CPV, and it consequently led to a disease of epizootic status with no prediliction for either sex or breed.
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Fluorescent Proteins for Neuronal Imaging,ion. Sites of virus multiplication are determined to a large extent by the fact that virus replication can only be supported by host cells which are actively dividing and so may be modified by any factor which influences the mitotic index of host tissue.
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