Titlebook: Cancer Prevention; Hans-J?rg Senn,Ursula Kapp Conference proceedings 2007 Springer-Verlag Berlin Heidelberg 2007 Carcinom.Genetic/biologic

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Genetic Risk Profiles for Cancer Susceptibility and Therapy Responsecies in host defense mechanisms against DNA damage (metabolic and DNA repair enzymes) can modify cancer susceptibility as well as therapy response. Genetic profiles should help to identify high-risk individuals who subsequently can be enrolled in preventive measures or treated by tailored therapy re

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Tumor Promotion as a Target of Cancer Preventioneoplastic and premalignant cells. It is caused by a continuous disturbance of cellular signal transduction that results in an over-stimulation of metabolic pathways along which mediators of cell proliferation and inflammation as well as genotoxic by-products are generated. Among such pathways the ox

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Insulin-Like Growth Factor-Related Signaling and Cancer Developmentisk assessment and prevention will be discussed. We reviewed background information related to IGF physiology at the cellular and whole-organism levels, together with prior work concerning IGF-I levels and risk of a variety of cancers, including breast, prostate, colon, and lung in 2004 (Pollak et a

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Prevention of Hepatocellular Carcinoma in Chronic Viral Hepatitis Patients with Cirrhosis by Carotenethods for its prevention. In the present study, the effect of a carotenoid mixture on hepatocellular carcinoma development was examined. Patients were randomly divided into two groups and treated with a carotenoid mixture in addition to conventional antisymptomatic treatment, or antisymptomatic tre

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Cox-2 and Cancer Chemoprevention: Picking up the Piecesnfortunately, these agents also cause toxicity in susceptible individuals. The recent reports of cardiovascular adverse events in patients treated with selective Cox-2 inhibitors for colorectal adenoma prevention remind us that all therapies carry risks as well as benefits. This article will discuss

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Human Papillomavirus Vaccine: A New Chance to Prevent Cervical Canceron in the United States. HPV is the primary etiologic agent of cervical cancer and dysplasia. Thus, cervical cancer and other HPV-associated malignancies might be prevented or treated by HPV vaccines. Recent research on the safety and efficacy of candidate prophylactic vaccines against HPV have show

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Prevention and Early Detection of Ovarian Cancer: Mission Impossible? 2,500) significantly affects strategies for prevention and detection. If chemoprevention for ovarian cancer were provided to all women over the age of 50, side effects would have to be minimal in order to achieve an acceptable ratio of benefit to risk. This ratio might be improved by identifying su

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Prevention of Prostate Cancer: More Questions than Datapidemiology of the disease gives some indications that its etiology is probably not only genetic but also environmental. Pathological studies support the fact that geographic differences in incidence and prevalence do not stem from genetic variations as men with the same genetic background raised in