Titlebook: Cancer Genes; Functional Aspects Enrico Mihich,David Housman Book 1996 Springer Science+Business Media New York 1996 apoptosis.cancer.cell.

Opponent

SIT

Isaac John,Santhakumar Mohan,Philippe Wengerween quiescent and growing states. This regulation relies on the coordinated balance between inhibitory and stimulatory signals. The stimulatory factors (e.g., growth factors) and their receptors are often encoded by a class of genes called proto-oncogenes. Cells also contain several inhibitory prot

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CULP

Efficiency Analysis of Shifted Spur Gears,sion cycle or Go reversible growth arrest. Here we consider the known biological activities of a set of genes named . (growth arrest specific) that are expressed during Go in NIH3T3 fibroblasts thus identifying an intrinsic genetic program involved in the maintenance of Go..The products of the . gen

Anal-Canal

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Zhaozhong Jiang,Garth M. Dahlen,Ayusman Senalignancies, leading to oncogenic activation either by the formation of chimeric transforming genes or by deregulation of transcription (Croce, 1987; Rabbitts, 1994). Gene fusion is observed in the majority of the myeloid tumors and in soft tissue sarcomas, and less frequently in B and T cell leukem

Ligament

Ali Benzaamia,Mohamed Ghrici,Redouane Rebouhncer drugs presently available to the clinic were initially discovered as substances that inhibit the proliferation of tumors in animal models and/or of cultured cells (see . for reviews). The field of cancer chemotherapy started with the serendipitous discovery that mustard gas agents used in chemi

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staging

Sandra Lagarto,Carlos A. Braumannanding of cytokine biology are the redundancy and pleiotropy of cytokine functions. Numerous in vitro systems suggest that many cytokines demonstrate overlapping activities, and that any individual cytokine demonstrates multiple activities. These findings, considered together with the complex circui

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