Titlebook: CNS Neuroprotection; Frank W. Marcoux (Vice President, Biology),Dennis Book 2002 Springer-Verlag Berlin Heidelberg 2002 Central Nervous S

SPARK

書目名稱CNS Neuroprotection影響因子(影響力)




書目名稱CNS Neuroprotection影響因子(影響力)學科排名




書目名稱CNS Neuroprotection網絡公開度




書目名稱CNS Neuroprotection網絡公開度學科排名




書目名稱CNS Neuroprotection被引頻次




書目名稱CNS Neuroprotection被引頻次學科排名




書目名稱CNS Neuroprotection年度引用




書目名稱CNS Neuroprotection年度引用學科排名




書目名稱CNS Neuroprotection讀者反饋




書目名稱CNS Neuroprotection讀者反饋學科排名

吝嗇性

議程

Our World is a Narrative World,n the patient’s family, the health care system, and society in general. The National Head and Spinal Cord Injury Survey estimates the rate of occurrence of severe head injury at 200 per 100000 (. et al. 1996). Motor vehicle accidents are the primary cause of both brain and spinal cord injury, and young healthy males are the primary victims.

挑剔小責

晚來的提名

Traumatic Central Nervous System Injuryn the patient’s family, the health care system, and society in general. The National Head and Spinal Cord Injury Survey estimates the rate of occurrence of severe head injury at 200 per 100000 (. et al. 1996). Motor vehicle accidents are the primary cause of both brain and spinal cord injury, and young healthy males are the primary victims.

FLIC

,Introduction Narrative’s Lost Body,m in this pathologic sequence has prompted the development of glutamate and calcium receptor antagonists. Unfortunately, these strategies have not produced in humans the degree of protection observed in animal models of acute necrotic stroke (. and . 1999).

FLIC

正面

Narrationen in der politischen Bildunging specificity for blocking Ca. and Na. channels have proven to be neuroprotective in various animal models. Several of these have been studied in placebo-controlled clinical trials. Unfortunately, to date, none of these compounds has unequivocally reduced human brain damage from stroke, cardiac arrest, or head trauma.

搖曳的微光

Sodium and Calcium Channel Blockersing specificity for blocking Ca. and Na. channels have proven to be neuroprotective in various animal models. Several of these have been studied in placebo-controlled clinical trials. Unfortunately, to date, none of these compounds has unequivocally reduced human brain damage from stroke, cardiac arrest, or head trauma.

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