Titlebook: CD95; Methods and Protocol Patrick Legembre Book 2017 Springer Science+Business Media LLC 2017 Cell motility.Immunoprecipitation.TNF Recept

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書(shū)目名稱(chēng)CD95影響因子(影響力)




書(shū)目名稱(chēng)CD95影響因子(影響力)學(xué)科排名




書(shū)目名稱(chēng)CD95網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱(chēng)CD95網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱(chēng)CD95被引頻次




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書(shū)目名稱(chēng)CD95年度引用




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書(shū)目名稱(chēng)CD95讀者反饋




書(shū)目名稱(chēng)CD95讀者反饋學(xué)科排名

造反,叛亂

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Immunoprecipitation of Death Inducing Signaling Complex by Caspase-8,ation is an elegant and efficient procedure to investigate endogenous protein interactions or complexes. Provided that the targeted complex is soluble in mild detergent these complexes can be recovered using protein A/G-coupled Sepharose beads and further analyzed after denaturation and electrophore

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In Vitro Evaluation of the Apoptosis Function in Human Activated T Cells, the intrinsic pathway on in vitro activated T cells. Her, we describe an in vitro assay allowing the monitoring of three different apoptosis pathways: (1) the FAS-induced pathway, (2) the activation-induced cell death (AICD), and (3) the death induced by starvation of the cells, called activated-ce

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Proximity Ligation Assay (PLA) to Evaluate DISC and MISC Composition,c signal at the detriment of the non-apoptotic signaling pathway [1–6]. Also CD95 can form different protein complexes via dynamic protein–protein interactions (PPIs) according to its interaction with soluble or transmembrane CD95L. Therefore, spatiotemporal identification of these PPIs is pivotal t

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CD95-Mediated Calcium Signaling,r calcium signals that control the cell fate, apoptosis, or survival, depending on the ligand (membrane or soluble). Intracellular calcium determination is an exquisite readout to explore the molecular mechanisms elicited by CD95 engagement. The most widely applied methods for studying calcium signa

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Study of the CD95-Mediated Non-apoptotic Signaling Pathway: PI3K,ence 245(4915):301–305, 1989). Accumulating evidence indicate that this so-called death receptor can also trigger non-apoptotic signaling pathways promoting inflammation and oncogenesis (Barnhart et al., Embo J 23(15):3175–3185, 2004; Chen et al., Nature 465(7297):492-496, 2010; Legembre et al., Cel