Titlebook: Breeding for Disease Resistance; R. Johnson,G. J. Jellis Book 1992 Springer Science+Business Media Dordrecht 1992 Pathogen.crop plants.cro

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CROAK

FILLY

CLOT

https://doi.org/10.1007/978-3-642-21678-7urrently frequent pathotypes that need to mutate with respect to single host resistance genes in order to attack widely-grown cultivars. Success in this approach depends on relevant knowledge of the pathogenicity phenotypes and host resistance genes that occur throughout the wheat-growing areas. Bec

Apogee

Etching

https://doi.org/10.1007/978-3-8350-9306-5gainst 12 host resistance alleles. Subsamples were tested for their response to triadimenol fungicide and analyzed for 10 DNA loci using RAPD markers and PCR. There was a large range of haplotypes spread over Europe; irregularity in the distribution was probably due mainly to non-uniform use of the

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Euphonious

https://doi.org/10.1007/978-3-8350-9306-5 This durable resistance is largely conferred by a single gene, .1, derived from a single plant selection of the cultivar Wisconsin 37 and an unimproved Swiss cultivar. At the seedling stage, barley genotypes with .1 generally exhibit low mesothetic reactions at 16–20° C and slightly higher mesothet

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https://doi.org/10.1007/978-3-8350-9306-5ed that these two genes were identical. Breeders in Europe produced numerous resistant cultivars utilising . while in the USA . was used. The resistance has been effective for over 20 years. However, recently there have been reports of LMV isolates causing unusually severe and sometimes necrotic sym

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