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Titlebook: Breathing, Feeding, and Neuroprotection; Ikuo Homma (Professor and Chairman),Seiji Shioda ( Conference proceedings 2006 Springer-Verlag To

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發(fā)表于 2025-3-21 16:04:08 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Breathing, Feeding, and Neuroprotection
影響因子2023Ikuo Homma (Professor and Chairman),Seiji Shioda (
視頻videohttp://file.papertrans.cn/191/190644/190644.mp4
發(fā)行地址Prior works in respiratory pattern generation
圖書封面Titlebook: Breathing, Feeding, and Neuroprotection;  Ikuo Homma (Professor and Chairman),Seiji Shioda ( Conference proceedings 2006 Springer-Verlag To
影響因子.New findings in brain research are being revealed on an almost daily basis, and the focus of this book is the fields of breathing, neuroprotection, and higher brain functions. An unresolved issue within respiration research and hence a topic of much interest is Where and how respiratory rhythm is generated in the brainstem, detailed analysis of which is presented herein. Chapters on neuroprotection examine the functional significance of the blood – brain barrier as an interface of blood and the central nervous system; other chapters look at health and disease in relation to the hypothalamic and limbic systems. In addition to animal experiments, research on the human brain is included, with a focus on the recently developed EEG/dipole tracing method. This book will be an invaluable reference for researchers in neuroscience and related fields..
Pindex Conference proceedings 2006
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The Role of the Blood-Brain Barrier in Feeding: Leptinentral nervous system (CNS) fluids (the brain interstitial and the cerebrospinal fluid). As such, it is a pivotal point in a humoral-based, endocrine-like communication between the CNS and peripheral tissues. For example, the BBB controls the entry of major feeding hormones into the CNS from the blo
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Ultrastructural Study on the Demyelination of the White Matter in the Rat Spinal Cord after Cardiac ly we reported that the neuronal damages in the hippocampus and lumbar spinal cord were induced after the combination of cardiac arrest and massive hemorrhagic shock. In the model, the neurons in intermediate gray matter were showed necrotic neuronal cell death at 1 d after ischemia. However, the da
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Engagement of Proinflammatory Cytokines after Cerebral Ischemiaf neuronal cell death after stroke and develop the new strategy of therapy, several studies had been performed during the past decade and some proinflammatory cytokines were identified as the therapeutic candidate. However, the critical actions of proinflammatory cytokines are still largely unknown
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Levels of the Alkoxy Radical in Patients with Brain Deathherapeutic strategy for treating or preventing neuronal damage. However, the study of free radicals in body fluids is currently severely hampered by technical difficulties in their detection. We have developed an ex vivo electron spin resonance (ESR) method, that employs 5,5-dimethyl-1-pyrroline-N-o
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