Titlebook: Brain and Heart Infarct II; K. J. Zülch,W. Kaufmann,V. Hossmann Conference proceedings 1979 Springer-Verlag Berlin Heidelberg 1979 brain.c

清楚

Figate

ADAGE

螢火蟲

Infusion

Ultrastructural and Biochemical Evidence for Reversibility of Regional Cerebral Ischemia,nditions are frequently the result of a coronary-artery occlusion that may be complicated by global ischemia due to cardiac arrest. Ischemic situations affecting the brain may be created through a variety of mechanisms that range from an extrinsic arterial occlusion through intrinsic venous thrombos

四牛在彎曲

Circadian Variations of Hemodynamics and Stroke,(24, 31), increased platelet adhesiveness (8), and aggregation (1) may combine with hemodynamic factors to impair local blood flow. The key factor provoking cerebral infarct, however, is atherosclerosis of the extra- or intracranial arteries, its localization being the determinant for the final neur

Malleable

Significance of Local Tissue PO2 and of Extracellular Cations for Functional and Reactive Hyperemia able to show with the aid of the local hydrogen clearance method (11) that the . in the brain cortex is also adapted to the metabolic demand during activation of the brain cortex. Fig. 1 shows an original registration of the microflow at four measuring sites in the superficial layers during direct

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Sergey Tulyakov,Venu Govindarajuause I am always dissatisfied with meetings in great hotels in cities, where we rush to and from the meeting rooms. I had such an experience overseas just 3 weeks ago from 8 o’clock in the morning until 10 o’clock at night.

Heart-Attack

Machine Learning in Educational Sciencesnce, by the World Federation of Neurology (7) on the basis of which it has directed corresponding world-wide research programs on the epidemiology of cerebrovascular disease (2, 6). We have made similar studies in our institute by measuring the lumina of the arterial stenoses (1, 9; Figs. 10–14).

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https://doi.org/10.1007/978-3-030-41068-1ently associated with extensive brain edema and in stroke where the tentorial herniation associated with the swelling that follows acute infarction is the usual cause of death (9). Increases in intracranial pressure and in hemispheral brain weight following middle cerebral occlusion have been reported (5).