Titlebook: Brain Repair; Anders Bj?rklund,Albert J. Aguayo,David Ottoson Textbook 1990Latest edition Macmillan Publishers Limited 1990 cells.central

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Functions of NGF on Central Cholinergic NeuronsNGFr content (total protein; Eckenstcin, 1988; Yan and Johnson, 1988), NGFr mRNA (Buck et al. 1987), total NGF content, but not NGF mRNA (Whittemore et al. 1986), rise during embryonic and neonatal basal forebrain development, suggesting that these regions might be preparing to contact a source of,

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Myelin-associated Inhibitory Substrate Components: Role in CNS Regeneration for this lacking elongation of lesioned CNS tracts (Ramon y Cajal, 1928) has become questionable with the finding that neurons cultured under optimal trophic factor conditions still did not produce processes into optic nerve expiants (Schwab and Thoenen, 1985). In the very same cultures, sciatic ne

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Axonal Pathfinding in the Developing Spinal Cord: Involvement of the Floor Plate in Chemotropic and Mendelson, 1986; Kuwada, 1986). Marked changes in the morphology of growth cones have been observed during the migration of axons through different cellular environments (Tosney and Landmesser, 1985), raising the possibility that growth cones may actively seek out such guidance cues.

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Logic-Based Incremental Process Minings in target cells is regulated by an, as yet unknown, exogenous factor (Rohrer et al., 1988). Retrograde flow of NGF, once established, persists for the life time of the particular neuron and is one of the factors which controls the survival and/or the maintenance of the differentiated state of the cell.

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Nonparametric Bayesian Deep Visualizationcortical grafts placed into frontal cortical lesions made in the .. Our analysis of the relatively discrete thalamocortical projection system in this experimental paradigm is compared to the trophic effects exerted on the more diffuse cholinergic projection system by neocortical transplants placed into . host cortical lesion cavities.