Titlebook: Brain Ischemia; Eugene Gusev,Veronica I. Skvortsova Book 2003 Springer Science+Business Media New York 2003 brain.death.depression.glutama

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Delayed Neuronal Death Following Acute Focal Brain IschemiaThe major part of a brain infarction is formed within the first 3–6 h of the first clinical signs of strake (Fig. 6.1). However, the temporal evolution of infarction and its “up-formation” are connected with secondary brain cell damage which takes place in hours to days after the initial vascular event.

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Programmed Cell Death. Apoptosis in Focal Brain IschemiaUntil the 1980s it was believed that neuronal death in acute brain ischemia occurred only by the mechanism of necrosis. However, later studies elucidated the role of apoptosis, another pathway to infarction, which is a variant of programmed cell death.

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Molecular Mechanisms of Post-Ischemic Reparation EventsWhen effective blood flow is recovered before irreversible necrotic damage forms, a stereotype reparative process will have been started in the brain tissue.

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Modern Therapeutic Approaches to Acute Focal Brain Ischemia. Basic Strategies for NeuroprotectionModern scientific and technological advances give reason to revise the previous pessimistic opinion about the therapeutic possibilities for acute focal brain ischemia, i.e. ischemic stroke.

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Lisa French. “Phototherapy” 100 years ago. The first American treatise on the uses of blue light. thought of light as a mode of medical treatment and ignored its use in the management of jaundice in the newborn infant, a condition which, if unchecked, can be crippling to a severe degree.

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