Titlebook: Brain Injury; Robert S. B. Clark,Patrick Kochanek Book 2001 Springer Science+Business Media New York 2001 Alzheimer.Neurointensiv.Trauma.a

ELUDE

Lucio Biasiori,Giuseppe Marcoccith beneficial and detrimental effects of these drugs. Evidence from both animal and human investigations now allows reasonable scientific basis for use of these compounds in humans suffering acute brain injury.

Projection

Apoptosis

Management of Emergency Situations,l perfusion pressure is critical for the effective treatment of head-injured patients, an important additional therapeutic strategy would be to restore the vasodilatory and vasoconstrictory properties of the cerebral vasculature that are intended to protect the brain by maintaining as constant and ideal an environment as possible.

打包

反感

Petra H?rn,Frank Günther,Simon Dückert however, the complexity of these interactions suggests we still have much to learn. This review will focus on recent insights into how interactions between trophic and neurotransmitter signals may participate in neuronal survival after TBI.

啞劇

拋物線

Molecular and Cellular Mechanisms of Traumatic Cerebral Vascular Injury,l perfusion pressure is critical for the effective treatment of head-injured patients, an important additional therapeutic strategy would be to restore the vasodilatory and vasoconstrictory properties of the cerebral vasculature that are intended to protect the brain by maintaining as constant and ideal an environment as possible.

engender

吼叫

Cell Signaling: Serine/Threonine Protein Kinases and Traumatic Brain Injury, however, the complexity of these interactions suggests we still have much to learn. This review will focus on recent insights into how interactions between trophic and neurotransmitter signals may participate in neuronal survival after TBI.

engagement

Neurotransmitter and Growth Factor Alterations in Functional Deficits and Recovery Following Traumargic systems as prototypic neurotransmitter systems for memory and frontal lobe function. Neuronal survival and/or regeneration may be influenced by changes in growth factors following TBI. Therefore, this chapter will also review studies of growth factor responses to TBI and the therapeutic efficacy of exogeneously administered growth factors.