Titlebook: Brain Edema IX; Proceedings of the N Umeo Ito (Director and Neurosurgeon in Chief),Alex Conference proceedings 1994 Springer-Verlag/Wien 19

Locale

https://doi.org/10.1007/0-387-31804-6nses to the disease. We have developed a safe, easily used rat model for . type b meningitis. We measured cerebral blood flow, glucose utilisation and second messenger activity in this model, and carried out parallel light and ultrastructural analysis of glial and neuronal responses. Only protein ki

本土

假設(shè)

SENT

Valeria Panebianco,Jurgen J. Fütterer minutes of 2 mM glutamate, with or without glutamate antagonists (dizocilpine, kynurenate, or NBQX), were allowed to recover for 60 minutes. The protocol was identical to that in noninvasive multinuclear NMR spectroscopy studies (.P/.H/.F) of live slices. Percent water and swelling were determined

藝術(shù)

venous-leak

NOT

報復(fù)

978-3-7091-9336-5Springer-Verlag/Wien 1994

ingestion

Acta Neurochirurgica Supplementhttp://image.papertrans.cn/b/image/190161.jpg

mucous-membrane

Neuron-Glial Interaction During Injury and Edema of the CNSators are glutamate, acidosis, free fatty acids, or high extracellular potassium. Glial homeostatic mechanisms are activated to prevent the secondary injury from these mediators. The glial clearance mechanisms have been studied in detail using in vitro systems allowing for a close control of the gli