Titlebook: Brain Edema / Cerebello Pontine Angle Tumors; Pathophysiology and K. Schürmann,M. Brock,D. Voth Conference proceedings 1973 Springer-Verla

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Nature and Significance of the CUSHING Reflexresponse. Two questions remained controversial since then: the adequate stimulus for the APR (WEINSTEIN, LANGFITT, CASSELL, 1964) and the significance of the APR in intracranial hypertension. It was described by SCHUTTA, KASSELL, LANGFITT (1968) and others that arterial hypertension may enhance the

Epidural-Space

CHURL

Intracranial Pressure, Hemodynamics and Metabolic Disorders in Patients with Severe Head Injury to about 40–60 mm Hg without marked cerebral hemodynamic or metabolic disorders (1, 2, 3). Below this limit significant decrease of cerebral blood flow and increase of cerebral tissue and jugular vein lactate concentration was noticed (4, 5, 6, 7, 8). In the above experiments low cerebral perfusion

冒失

Hypertonic Solutions and Diuretics in the Treatment of Brain Edeman as a new presumably more potent drug is discovered. Since decades this is the general practice, a practice molded by clinical observations and experimental results as well as theoretical concepts, analogies and traditions.

Ingenuity

Electroencephalographical Findings During Therapy of Brain Edema with Dexamethasoneent for cerebral edema (5, 6, 7, 10). At the same time they discussed several interesting problems. In order to gather more clinical information in man, we performed an electroencephalographic observation on the course before and during treatment with dexamethasone.

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Influence of Ventilation and Hyperventilation on Brain Edema and Intracranial Pressurefficult from the neurosurgical point of view. There seem to be only few exact studies that are apt for clinical interpretation. For this reason, it seems necessary to accept the cerebrospinal fluid pressure (CSF pressure) as a further factor of intracranial pressure. In the course of a lumbar tappin

BRAND

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周興旺

https://doi.org/10.1007/3-540-28746-9 if they persist for some time they are soon followed by vascular lesions. The capillary lumen may be narrowed by endothelial swelling. Midbrain hemorrhages occur when brain edema develops to brain death. — The following experiments were undertaken to clarify the mechanism of both phenomena.

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