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Titlebook: Brain Edema / Cerebello Pontine Angle Tumors; Pathophysiology and K. Schürmann,M. Brock,D. Voth Conference proceedings 1973 Springer-Verla

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發(fā)表于 2025-3-21 20:03:22 | 只看該作者 |倒序瀏覽 |閱讀模式
期刊全稱Brain Edema / Cerebello Pontine Angle Tumors
期刊簡稱Pathophysiology and
影響因子2023K. Schürmann,M. Brock,D. Voth
視頻videohttp://file.papertrans.cn/191/190160/190160.mp4
學(xué)科分類Advances in Neurosurgery
圖書封面Titlebook: Brain Edema / Cerebello Pontine Angle Tumors; Pathophysiology and  K. Schürmann,M. Brock,D. Voth Conference proceedings 1973 Springer-Verla
影響因子This volume contains the papers presented at the 24th Annual Meeting of the Deutsche Gesellschaft fur Neurochirurgie, held in Mainz, Western Germany, on April 30 - May 3, 1973. Deliberate choice was made of two crucial still hotly debated subjects which, for ages, have meant a source of constant worry, and nights without sleep to every neurosurgeon. Just as long as our special field exists, there have been the problems of how to control brain edema and ·of how to reduce lethality and the secondary lesions in surgery of cerebello-pontine angle tumors. Concerning the first subject, new pathological, pathophysiological and chemical aspects, the mechanisms of brain edema formation and resolution are presented in the hope for better understanding. Furthermore, the relationship between brain edema, intracranial pressure, cerebral blood flow and metabolism are discussed. Finally, the therapeutical consequences as well as the results of experimental and clinical work are presented, and a comparison of effects between different methods (hypertonic solutions, diuretics, steroids, controlled hyperventilation, hyperbaric oxygen) is given. Concerning the second main subject, any important contr
Pindex Conference proceedings 1973
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,MDA — überblick und Orientierung,blood-brain barrier is a cause of scintigraphic visualization of a brain lesion. However, the problem in brain tumors is quite different from that in brain contusion. It should be remembered that in tumors we are dealing with non-cerebral tissue. Their visualization in fact is dependent on the prese
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Soft Organ MDCT Imaging: Pancreas and Spleen are therefore frequently concerned to recognise whether ICP is raised, and if so to try to reduce the level of pressure. Continuous clinical monitoring of pressure was shown to be feasible by GUILLAUME and JANNY in France in 1951, and a large series of neurosurgical cases was reported from Sweden i
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Imaging Protocols for Cardiac CTior temporal extradural haematoma was removed. As she did not regain consciousness a check angiogram was done, and this showed some haematoma remaining. To help us decide whether a second operation was necessary, a ventricular fluid pressure recording was started on April 28th. It showed a basic pre
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Avinash R. Kambadakone,Dushyant V. Sahani to about 40–60 mm Hg without marked cerebral hemodynamic or metabolic disorders (1, 2, 3). Below this limit significant decrease of cerebral blood flow and increase of cerebral tissue and jugular vein lactate concentration was noticed (4, 5, 6, 7, 8). In the above experiments low cerebral perfusion
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https://doi.org/10.1007/978-88-470-1878-5n as a new presumably more potent drug is discovered. Since decades this is the general practice, a practice molded by clinical observations and experimental results as well as theoretical concepts, analogies and traditions.
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https://doi.org/10.1007/978-88-470-2403-8ent for cerebral edema (5, 6, 7, 10). At the same time they discussed several interesting problems. In order to gather more clinical information in man, we performed an electroencephalographic observation on the course before and during treatment with dexamethasone.
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