Titlebook: Bovine Spongiform Encephalopathy; The BSE Dilemma Clarence J. Gibbs Book 1996 Springer-Verlag New York, Inc. 1996 BSE.biochemistry.genetics

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https://doi.org/10.1007/978-1-4612-2406-8BSE; biochemistry; genetics; infection; infections; molecular biology; molecular genetics; pathogenesis

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Fortify

,übergang: Therapieabschluss und Nachsorge, In November 1986 the first cases of BSE were confirmed by histologic examination of the brain from affected cattle from two areas of southern England (2). Subsequently it was reported that clinical cases of BSE, unrecognized at the time, had probably occurred as early as April 1985 and that the ori

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Monocle

https://doi.org/10.1007/978-3-540-89226-7itial epidemiologic studies provided evidence that cattle had become infected by a scrapie-like agent via infected meat and bone meal used as a protein supplement (2). A subsequent case-control study of calf-feeding practices substantiated this hypothesis (3), and action to prevent further exposure

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,übergang: Therapieabschluss und Nachsorge, from animal-to-animal and flock-to-flock has been a subject of controversy, particularly whether transmission is vertical or horizontal, or both. The significant aspects of research efforts to define and evaluate the nature of vertical and horizontal transmission are presented. These include diagno

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Beziehungen und soziale Kompetenzen,accidentally contaminated by a scrapie-like agent (1). However, there remain several important questions related to the origin and biologic properties of BSE. First, what is the origin of the agent? Is it a previously unrecognized strain of BSE agent rarely inducing sporadic disease in cattle and ar

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Beziehungen und soziale Kompetenzen,ces of Wisconsin mink producers were conducted to estimate the maximum prevalence and incidence of a hypothetical bovine transmissible encephalopathy within the context of the following hypothesis: the source of agent for each TME outbreak in Wisconsin was food-borne exposure of mink to a cow with a