Titlebook: Blistering Diseases; Clinical Features, P Dédée F. Murrell Book 2015 Springer-Verlag Berlin Heidelberg 2015 Epidermolysis bullosa.Immunoflu

digestive-tract

書目名稱Blistering Diseases影響因子(影響力)




書目名稱Blistering Diseases影響因子(影響力)學科排名




書目名稱Blistering Diseases網絡公開度




書目名稱Blistering Diseases網絡公開度學科排名




書目名稱Blistering Diseases被引頻次




書目名稱Blistering Diseases被引頻次學科排名




書目名稱Blistering Diseases年度引用




書目名稱Blistering Diseases年度引用學科排名




書目名稱Blistering Diseases讀者反饋




書目名稱Blistering Diseases讀者反饋學科排名

CRAMP

The Immune System in Normal Skin and in AIBDnment, it protects against hazardous substances, such as chemicals and physical stress. Various immune cells reside in the two main compartments of the skin, the epidermis and the dermis, under physiological steady-state conditions. It is essential for maintaining the integrity of the skin that immu

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Desmosomal Proteins as Autoantigens in Pemphigusere are several variants of pemphigus, each with unique clinical, histological, and immunologic features. Interestingly, when different desmosomal proteins are targeted by the autoimmune response, different clinical and histological features are seen. In this chapter we review the key intercellular

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Collagen XVII and Its Role in Junctional Epidermolysis Bullosathelial cells to the underlying basement membrane. It is a homotrimeric type II transmembrane protein consisting of three 180 kDa alpha-1 (XVII) chains. Each individual chain is encoded by the . gene. Mutations in . usually lead to loss of collagen XVII and a phenotype comprising congenital generali

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Kindlin-1 and Its Role in Kindler Syndromedhesions, which serve as supramolecular signalling and actin-anchoring platforms. Together with talin, kindlins directly bind to integrin β subunit cytoplasmic tails and are responsible for integrin activation. Mutations in the kindlin-1 gene cause the Kindler syndrome, a rare genodermatosis charact