Titlebook: Blaustein‘s Pathology of the Female Genital Tract; Robert J. Kurman,Lora Hedrick Ellenson,Brigitte M. Living reference work 20200th editio

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https://doi.org/10.1007/978-3-662-25398-4nd pathogenesis. The World Health Organization classification of GTD includes complete and partial hydatidiform mole, invasive mole, choriocarcinoma, placental site trophoblastic tumor (PSTT), epithelioid trophoblastic tumor, exaggerated placental site, and placental site nodule (Table .). Some of t

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Verwendung des Eisens zu Decken, myeloid sarcomas and myeloid leukemias, and rarely histiocytic neoplasms. When the female reproductive organs are affected by one of these disorders in the setting of widespread disease, the diagnosis is usually straightforward, but in the uncommon instance in which the female genital tract is the

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Verwendung des Eisens zu Decken,ve significant adverse consequences for the patient. They may spread from adjacent sites by direct local extension or from distant extragenital sites. The latter tumors are truly metastatic, whereas the designation “metastatic” is sometimes not used for those that are secondary from local sites. For

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https://doi.org/10.1007/978-3-662-25398-4cking an ovarian neoplasm on clinical examination, at operation, or on pathologic examination. Many occur in the reproductive years and may be associated with infertility. Their proper recognition is, therefore, important to allow appropriate, usually conservative therapy, thereby avoiding unnecessa

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Verwendung des Eisens zu Decken,s location, occurring much more frequently in other parts of the body. Consequently, whenever they are encountered in the ovary, these tumors pose difficult problems in diagnosis, histogenesis, behavior, and therapy for the pathologist and clinician. These neoplasms must be differentiated from prima

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,Berechnung von Fachwerktr?gern, 20 years, it has been evident that specific types of human papillomavirus (HPV) cause almost all squamous cell carcinomas of the cervix as well as the vast majority of adenocarcinomas of the cervix. However, much remains to be learned regarding the precise molecular pathways by which these DNA tumo

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https://doi.org/10.1007/978-3-662-25398-4cycles, and exogenous hormones are associated with both endometrioid carcinoma and hyperplasia. In addition, the risk of endometrial hyperplasia is associated with increasing body mass index and nulliparity (Epplein et al. 2008; Wise et al. 2016; Guraslan et al. 2016). All of these factors are thoug