Titlebook: Bipolar Disorder: From Neuroscience to Treatment; Allan H. Young,Mario F. Juruena Book 2021 Springer Nature Switzerland AG 2021 Bipolar.Tr

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Bipolar Disorder: From Neuroscience to Treatment978-3-030-72143-5Series ISSN 1866-3370 Series E-ISSN 1866-3389

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Juridicum – Schriftenreihe zum Strafrechtormones, inflammation, oxidative stress and neurotrophins are altered in bipolar disorders, especially during acute mood episodes. Together, these changes have been associated with a systemic toxicity of the disease and the damage resulting from multiple episodes. Systemic toxicity related to recurr

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https://doi.org/10.1007/978-3-658-28040-6eover, patients who have bipolar disorder generally show impaired brain function, including reward processing. There is some evidence for a causal role of (dysfunction of) the stress system in the etiology of bipolar disorder and their effects on brain system functionality. However, longitudinal stu

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https://doi.org/10.1007/978-3-658-16036-4on of rare variants, which may be responsible for the relatively low heritability explained (~20% in the largest GWAS) compared to twin studies. The availability of data from large biobanks and automated phenotyping from electronic health records or digital phenotyping represent key steps for provid

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Legitimit?t und politische Machtmmune pathways are often associated with BD pathophysiology. Hence, in this chapter, we focus on the evidence for the involvement of inflammation and immune regulated pathways in the neurobiological consequences of BD neuroprogression. Herein we put forth the evidence of immune markers from autoimmu