Titlebook: Biomechanics and Mechanobiology of Aneurysms; Tim McGloughlin Book 2011 Springer-Verlag Berlin Heidelberg 2011 abdominal aorta.aetiology.a

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https://doi.org/10.1007/978-3-663-10535-0ic aneurysm do not exhibit some of the native characteristics of the disease and as such are not suitable for investigating disease initiation and progression. The current review summarizes the various approaches of animal models for aortic aneurysm in the context of their appropriateness for biomec

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1868-2006 de new insights into aneurysm aetiology and behavior based on the most recent biomechanics research related to this important topic. The contributors to this volume br978-3-642-27075-8978-3-642-18095-8Series ISSN 1868-2006 Series E-ISSN 1868-2014

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Aneurysm: Epidemiology Aetiology and Pathophysiology,ies, atherosclerotic distribution, proteolytic pattern, and cell signaling pathways that have implications in the development of an aortic aneurysm. During the last decades an overwhelming amount of evidence has been accumulated in support of genetic risk factors contributing to the development, gro

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Biomechanics and Pathobiology of Aortic Aneurysms,c aneurysms arise from very different pathophysiologies that ultimately result in a final common outcome of matrix degeneration and biomechanical failure. Therefore, the patient-specific knowledge of both wall stress and wall strength distributions for a given aneurysm will greatly improve the abili

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Biomechanical Aspects of Abdominal Aortic Aneurysm (AAA) and its Risk of Rupture: Fluid Structure Iating that the latter may underestimate the AAA risk of rupture. The ILT appeared to provide a cushioning effect reducing the stresses, while small calcifications appeared to weaken the wall and contribute to the rupture risk. The location of the maximal wall stresses and rupture potential index (RP

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Computational Analysis of Displacement Forces Acting on Endografts Used to Treat Aortic Aneurysms,pendent on the tortuosity and size of the endograft, as well as on the hemodynamic state of the patient. The fixation response of the endograft is determined by factors such as the fixation mechanism (radial pressure vs. hooks and barbs), amount of “l(fā)anding zone” (i.e., area where the endograft can

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