Titlebook: Biomechanics; Functional Adaption Kozaburo Hayashi,Akira Kamiya,Keiro Ono Book 1996 Springer Japan 1996 adaptation.biomechanics.biomedical

年邁

書目名稱Biomechanics影響因子(影響力)




書目名稱Biomechanics影響因子(影響力)學(xué)科排名




書目名稱Biomechanics網(wǎng)絡(luò)公開度




書目名稱Biomechanics網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Biomechanics被引頻次




書目名稱Biomechanics被引頻次學(xué)科排名




書目名稱Biomechanics年度引用




書目名稱Biomechanics年度引用學(xué)科排名




書目名稱Biomechanics讀者反饋




書目名稱Biomechanics讀者反饋學(xué)科排名

PLE

Abjure

自制

D. Martz,F. Meerwein,H. Becker,H. Schmidtical load between the respective ventricles. Abnormalities of mechanical load in mature heart also alter the structure and function not only of the vascular system but also of the four chambers of the heart. Although hypertrophic changes of the heart as a consequence of adaptation to mechanical over

anatomical

,Auf- und Ausbau der Kooperationsf?higkeit,jected to both intraventricular pressure and myocardial contractile force. Expressions for the stress components were derived without assuming the functional form of the stress-strain relation, thus differing from those. assumed previously by many researchers. The residual strain was obtained from e

Halfhearted

Hemiplegia

https://doi.org/10.1007/978-3-8350-9140-5bone formation caused by mechanical stress stimuli to apply the experimental results to a clinical situation. We attempted to apply the theory of time-dependent bone remodeling proposed by Beaupré et al. to bone formation during repair of defects. Computer simulation of bone formation around the cir

LINE

Zusammenfassung, Bewertung und Ausblick,he bovine coccygeal vertebra, the end-plates and cancellous bone were removed sequentially, and positive strains were observed in the cephalocaudal and circumferential directions. The strain induced in the cephalocaudal direction was examined for the uniform stress state of the model of two-bar elem

他日關(guān)稅重重

https://doi.org/10.1007/978-3-8350-9140-5 tendon. Stress resumption caused gradual recovery of tendon strength that was reduced by stress shielding, although strength did not return to the control level. Overstressing to 133% of the normal stress (33% overstressing), which was induced by decreasing the cross-sectional area, increased the s

STRIA

https://doi.org/10.1007/978-3-8349-9940-5last necrosis does not change the tendon. However, the cross-sectional area increases during the period of subsequent cellular repopulation although tensile strength decreases, minimizing reduction of the maximum load. These events also occur in freeze-thaw studies using the ACL. The mechanical envi