Titlebook: Biology of Women’s Heart Health; Lorrie Kirshenbaum,Inna Rabinovich-Nikitin Book 2023 The Editor(s) (if applicable) and The Author(s), und

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Auswahl des richtigen Analgetikums,sed ventricular wall stiffness which is in turn manifest as the incidence of heart failure and cardiac fibrosis per se and is known to be a primary contributor to the occurrence of heart failure. In this chapter, we seek to explore possible signaling differences among males and females that might co

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Schmerztherapie mit Nicht-Analgetika,te premature development and progression of coronary artery disease in older premenopausal women reporting a history of hypothalamic ovarian disruption. Importantly, in women with EAA, estrogen therapy and resumption of menses restores endothelial function. In this review we focus on the influence o

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Preeclampsia: Early and Long-Term Clinical Considerations,e postpartum period, highlighting the need for post-partumassessment. Though much work has been done in the antepartum diagnosis and management of preeclampsia,a growing body of evidence has shown an increased risk of long-term cardiovascular disease in patients whodevelop preeclampsia. Not only mus

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Gestational Diabetes as a Risk Factor for Cardiovascular Disease,tegies. A diagnosis of GDM offers women, clinicians, and policy makers a unique opportunity to implement effective screening and treatment strategies to reduce cardiovascular disease in this population. Much more research is needed to identify the best evidence-based practices for cardiovascular dis

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Menopause and the Bridge to Cardiovascular Disease,biological hazards, as does a lack of education and training for women’s cardiovascular health. Estrogen replacement therapy offers some potential benefits for postmenopausal women but the knowledge gaps in our understanding of how estrogens impact cardiovascular health specifically, and postmenopau

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Cardiometabolic Function in Women, higher prevalence of cardiovascular events until mid-age. However, after menopause, the women-to-men ratio of cardiovascular events increases. Biological explanations for gender differences in cardiometabolic diseases are complex. The main physiological features that underlie the gender differences