Titlebook: Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation; Yuansheng Gao Book 20171st edition Springer Nature Singapore Pte Ltd.

力學(xué)

書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation影響因子(影響力)




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation影響因子(影響力)學(xué)科排名




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation網(wǎng)絡(luò)公開(kāi)度




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation網(wǎng)絡(luò)公開(kāi)度學(xué)科排名




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation被引頻次




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation被引頻次學(xué)科排名




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation年度引用




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation年度引用學(xué)科排名




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation讀者反饋




書(shū)目名稱(chēng)Biology of Vascular Smooth Muscle: Vasoconstriction and Dilatation讀者反饋學(xué)科排名

ARK

GONG

Rechenbeispiele in der Transfusionsmedizin,ity in the tension development when its length is altered and in the shortening speed when its load is changed. This is ascribed to a dynamic process of formation of cross-bridge by myosins and actins and lends VSM marked flexibility to meet different physiological needs. The above topic will be dis

STEER

Transfusionelle CMV-Problematik,oactive intestinal peptide, nitric oxide, calcitonin gene-related peptide, substance P, and others. Some of them are vasoconstrictors and some are vasodilators. The balance of the activities of these neurotransmitters is critical for the integrity of vascular function.

Malaise

https://doi.org/10.1007/978-3-211-89851-2sed Ca. influx. Among these vasodilators, NO is the dominant player, PGI. often has a complementary role, and EDHFs are more important in resistant arteries where NO is less crucial. The vasodilatory effects of these agents are counteracted by various vasoconstrictors in particular ET-1, which exert

AWE

萬(wàn)神殿

,Thrombozytopenischer Refrakt?rzustand,onse is initiated by the opening of mechanosensitive ion channels such as TRPC6 channel, TRPM4 channel, and probably a heteromultimeric channel complex formed by the β and γ subunits of epithelial sodium channels and the subunit 2 of acid-sensing ion channel. Myogenic response is of fundamental impo

annexation

Book 20171st editione responsiveness of blood vessels under normal and pathophysiological conditions help to provide valuable weapons in the fight vascular diseases. The book is of interest to researchers and graduate students, both in basic research and in clinic settings, in the field of vascular biology..

激勵(lì)

Architecture of the Blood Vesselswill be discussed. Furthermore, the gap junctions that form intercellular connections and the vessels that provide nutrient to vessel walls as well as some cell types of functional importance including fibroblasts, stem/progenitor cells, and perivascular adipose tissue will be reviewed.

CAGE

Vascular Endotheliumnhibitory actions on platelet aggregation and coagulation and its facilitating effect on fibrinolysis. These effects are mainly mediated by the endothelium-derived molecules such as nitric oxide, prostaglandin I., thrombomodulin, heparan sulfate proteoglycan, and tissue factor pathway inhibitor. How