Titlebook: Biology and Physiology of the Blood-Brain Barrier; Transport, Cellular Pierre-Olivier Couraud,Daniel Scherman Book 1996 Springer Science+B

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Distribution of Small Neutral Amino Acids after Penetrating the Luminal Side of the Guinea Pig Bloodease of volume of distribution in postvascular compartment (brain parenchyma), in comparison with the vascular compartment (pellet)..These results indicate that small neutral amino acids, after penetrating the luminal side of the blood-brain barrier, are probably accumulated in brain endothelial cel

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Vascular Permeability to Hemorphins in the Central Nervous Systemod. Infusion of hemorphin-7 however, did not influence the regional brain water content or ultrastructure of the cerebral microvessels compared to either radioactive iodine or Leu-Val-Val-hemorphin-7. These results suggest that hemorphin-7 has the capacity to cross the BBB of normal rats without aff

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The Effect of Glycosylation on the Uptake of an Enkephalin Analogue into the Central Nervous Systemer-gly. In summary, the difference in analgesic response of glycosylated compared to unglycosylated DCDCE-ser-gly, is not related to either differing metabolic profiles, nor the ability of the glycosylated analogue to use the glucose carrier to enter the CNS. However, this study does not eliminate t

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Development of Blood-Brain Barrier Tight Junctionshancement between stages E 18 and Pl. Particle insertion starts between E 13 and E 15 and shows predominant association to the extracellular fracture face until stage E18. At E13 almost no particles can be observed, neither on P-, nor on E-faces. Analogue results were obtained from Ca.-depleted cult

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https://doi.org/10.1007/978-3-322-99773-9al cell growth and differentiatian may be mediated by lyn, a nonreceptor tyrosine kinase expressed in brain endothelium. Induction and maintenance of blood-brain barrier endothelial cell characteristics (complex tight junctions, low number of vesicles, specialized transport systems) are regulated by

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Ralf Kleinfeld,Ralf Heidemann,Frank Treutleron the luminal membrane and not accessible in these experiments. Although plasma taurine was increased by hypo-osmotic stress in vivo, this was not reflected by an increase in taurine influx across the blood-brain barrier. Thus this barrier tissue also prevents taurine from being recycled back into

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Lipohypertrophy

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Stellung und Funktion der Kreises’ luminal membrane. Values for Michaelis-Menten constant for L-alanine transport from blood into brain point out that the affinity of this molecule to its carrier(s) is rather small (K. >1 mmol/1). Capacity of .H L-alanine blood-to-brain transport is very small as well (V. <20 nmol/min/g)..The addi