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Titlebook: Biological Responses in Cancer; Enrico Mihich Book 1985 Springer Science+Business Media New York 1985 angiogenesis.cancer.cell.chemotherap

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發(fā)表于 2025-3-21 18:10:44 | 只看該作者 |倒序?yàn)g覽 |閱讀模式
期刊全稱(chēng)Biological Responses in Cancer
影響因子2023Enrico Mihich
視頻videohttp://file.papertrans.cn/188/187454/187454.mp4
圖書(shū)封面Titlebook: Biological Responses in Cancer;  Enrico Mihich Book 1985 Springer Science+Business Media New York 1985 angiogenesis.cancer.cell.chemotherap
影響因子The series of volumes entitled Biological Responses in Cancer provides information on approaches through which the interaction between neoplas- tic and normal cells may be modified. Topics discussed in various volumes include immunological and host defense systems, control mechanisms of cell and population growth, cell differentiation, and cell transformation. This volume is specifically concerned with various aspects of cell interactions and regulation within heterogeneous tumor cell populations, and their role in tumor progression and metastasis. Knowledge in this area is likely to provide new leads toward the exploitation of novel cellular sites and mech- anisms in the development of new types of therapies of cancer. Several topics are discussed within these general areas of consideration. The possibly unique characteristics and mechanisms of tumor vascularization and the potential sites of interference with angiogenesis that might have therapeutic impli- cations are critically evaluated. Tumor cell-normal tissue interactions in- volved in different phases of the growth and metastatic processes are dis- cussed in two chapters dealing with mechanisms of tumor invasion and with th
Pindex Book 1985
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The Evolution of Phenotypic Diversity in Metastatic Tumor Cells,cted, heterogeneity also exists in their sensitivities to various therapeutic agents (such as drugs, radiation, and hyperthermia), as well as host—response mechanisms (reviewed in Hart and Fidler, 1981; Fidler and Hart, 1982; Nicolson, 1982, 1984a; Nicolson and Poste, 1982, 1983a,b). Heterogeneity i
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Cellular Heterogeneity of Human Tumors,e turned out to be more quantitative than qualitative (Markert, 1982; Nicolson, 1984a). Even more damaging to a simple “exploitable difference” approach is evidence, now accumulating, that significant differences may exist between malignant cells even within the same tumor. These differences may be
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Drainage des Pleuraspaltes — Pleurabiopsieifferentially stimulate synthesis of the lamina proteins, type IV collagen and laminin, have been detected in rodent and human mammary tumors. Most interestingly, the responsiveness of mammary cells to these growth factors is potentiated when the mammary cells interact with stromal collagen. This se
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Einteilungsprinzipien und Begriffe,cted, heterogeneity also exists in their sensitivities to various therapeutic agents (such as drugs, radiation, and hyperthermia), as well as host—response mechanisms (reviewed in Hart and Fidler, 1981; Fidler and Hart, 1982; Nicolson, 1982, 1984a; Nicolson and Poste, 1982, 1983a,b). Heterogeneity i
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H. K. Breddin,Th. Karasch,H. Riegerne dependence, antigenic expression, metastatic potential, and stem cell karyotype. With the availability of more sensitive markers, coupled with the refinement of detection techniques, an even greater genetic diversity of tumor cells is likely to be discovered in the future. However, knowledge as t
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Allgemeine Diagnose- und Therapiestrategien,ystems under controlled conditions, especially with murine tissue, the mouse mammary gland has proven to be an excellent model system for studying hormonal control of differentiation and preneoplasia.
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發(fā)表于 2025-3-23 06:32:17 | 只看該作者
,Invasive lumener?ffnende Therapie,e turned out to be more quantitative than qualitative (Markert, 1982; Nicolson, 1984a). Even more damaging to a simple “exploitable difference” approach is evidence, now accumulating, that significant differences may exist between malignant cells even within the same tumor. These differences may be
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