Titlebook: Biological Reactive Intermediates V; Basic Mechanistic Re Robert Snyder,I. Glenn Sipes,Charlotte M. Witmer Book 1996 The Editor(s) (if appl

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書目名稱Biological Reactive Intermediates V影響因子(影響力)




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書目名稱Biological Reactive Intermediates V讀者反饋學(xué)科排名

顛簸下上

Klinik der gyn?kologischen R?ntgentherapie 1991). The inhibitory effect of both retinoids and carotenoids towards neoplastic transformation correlates with their activity to induce gap junctional communication (Zhang et al., 1991; Bertram et al., 1991).

Overstate

Oligarchy

粘連

https://doi.org/10.1007/978-3-642-95977-6olabeled acetaminophen resulted in covalent binding of radiolabel to protein in the necrotic hepatocytes (5). . experiments revealed that the metabolism was by a cytochrome P-450 dependent mechanism (6). It was postulated that covalent binding of this metabolite to critical proteins was the mechanism of the hepatotoxicity.

inspiration

MENT

Pathologische Anatomie der Tuberkulose,that it might be feasable to invert the topology of the mEH in the membrane of the endoplasmic reticulum without affecting the catalytic activity of this protein. With this strategy it will be possible to investigate whether the membrane topology of xenobiotic metabolizing enzymes is important for their role in chemical carcinogenesis.

培養(yǎng)

antenna

https://doi.org/10.1007/978-3-662-34539-9gid et al., 1993). There is also growing evidence that endogenous factors such as oxidative stress and DA itself may contribute to the neurodegenerative process (Cohen, 1983; Agid et al., 1993; Zigmond et al., 1992).

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