Titlebook: Biological Reactive Intermediates III; Mechanisms of Action James J. Kocsis,David J. Jollow,Robert Snyder Book 1986 The Editor(s) (if appli

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Biological Reactive Intermediates III978-1-4684-5134-4Series ISSN 0065-2598 Series E-ISSN 2214-8019

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https://doi.org/10.1007/978-3-658-20573-7ransformation to chemically reactive species which covalently modify cellular macromolecules. Ten years ago.’. we proposed that bay-region diol epoxides are prime candidates for the ultimate carcinogenic metabolites of the carcinogenic hydrocarbons provided that these molecules contained a bay-regio

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Klimazeugnisse der Erdgeschichteof chemicals are mediated by reactive products formed during their biotransformation in the organism. It is equally clear that there exist a number of protective systems which can trap, or inactivate, toxic metabolites and thereby prevent their accumulation within the tissues and subsequent toxic ef

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Wolfgang Janke,Heinz Kliewe,Horst Sterre of their critical location along the sinusoids where they are the first cells to encounter blood borne xenobiotics. To study the possible role of the NPC in the metabolism of xenobiotics, populations of NPC and parenchymal cells (PC) were prepared from rats and various xenobiotic metabolizing enzy

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Das ?Wetter“ im Meer: Nord- und Ostseets that can be attacked by cellular nucleophiles or solvolyzed (Guengerich and Liebler, 1985). Much has been learned about the isozymes in this family of hemoproteins from studies with microorganisms and experimental animals. Biochemical studies with humans were started shortly after the discovery o

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