Titlebook: Biological Basis of Alcohol-Induced Cancer; Vasilis Vasiliou,Samir Zakhari,Jan B. Hoek Conference proceedings 2015 Springer International

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,Synergistic Toxic Interactions Between CYP2E1, LPS/TNFα, and JNK/p38 MAP Kinase and Their Implicatiress. Considerable attention has been given to alcohol-elevated production of lipopolysaccharide (LPS) and TNFα and to alcohol induction of CYP2E1. These two pathways are not exclusive of each other; however, associations and interactions between them, especially in vivo, have not been extensively e

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,Alcohol Consumption, Wnt/β-Catenin Signaling, and Hepatocarcinogenesis,sted that ethanol (EtOH) metabolism may enhance tumor progression by increasing hepatocyte proliferation. To test this hypothesis, ethanol (EtOH) feeding of male mice began 7 weeks post-injection of the chemical carcinogen diethylnitrosamine (DEN), and continued for 16 weeks, with a final EtOH conce