Titlebook: Biochemistry of Signal Transduction in Myocardium; Jos M. J. Lamers,Pieter D. Verdouw Book 1996 Kluwer Academic Publishers 1996 Angiotensi

含糊

catagen

不來

Angiotensin II signalling pathways in cardiac fibroblasts: Conventional versus novel mechanisms in mnalling pathways in common with those of peptide growth factors. Below, we describe growth-related aspects of cardiac fibroblasts with respect to angiotensin II receptors, conventional and novel signal transduction systems, secretion of extracellular matrix proteins and growth factors, and localizat

虛假

cleaver

Regulation of G protein function: Implications for heart diseasemyopathies are associated with abnormal GPCR function, mutations are unlikely in these disorders. The last few years, other aspects of G protein function have moved into focus: e.g. posttranslational modifications; effector regulation by . subunits; GTPase activating protein (GAP) activity of effect

MAPLE

Regulation and functional significance of phospholipase D in myocardium by agonists in myocardium is controversial. Agonists, such as endothelin-1, atrial natriuretic factor and angiotensin II that are shown to activate phospholipase D, also potently stimulate phospholipase C-β in myocardium. PMA stimulation of protein kinase C inactivates phospholipase C and strongly

主講人

Phosphotransfer reactions as a means of G protein activationof G protein-coupled receptors for agonists and an increased response of G protein coupled effectors. In addition, a phosphorylation of G protein β subunits and consequent phosphate transfer reaction resulting in G protein activation has also been demonstrated. Finally, endogenously formed GTP was p

針葉類的樹

Calibrate

Infraction