Titlebook: Biochemistry of Schizophrenia and Addiction; In Search of a Commo Gwynneth Hemmings (Honorary Secretary) Book 1980 MTP Press Limited 1980 b

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原來

https://doi.org/10.1007/978-3-322-96467-0zophrenic patients studied. This could mean that there are two groups of patients, one with an increased permeability for gliadin and with a high antigliadin titre, the other with a low permeability and having a low titre. This phenomenon is similar to that described in Chapter 13 by Ashkenazi .., i

異端邪說2

separate

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The relevance of immunopathology to research into schizophreniahat this discipline will prove decisive in terms of the techniques that are available now. However, there are three ways in which immunology may help us to understand the pathogenesis of schizophrenia by providing additional means of testing hypotheses proposed on other grounds.

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串通

Binding of chlorpromazine and HLA-A1 antibodies to human lymphocyte membranesinked to HLA gene products which might influence their interaction with CPZ. An alternative explanation offered was that there is perhaps some analogy between the structures of HLA-A1 antigens and the β-adrenergic receptors. Therefore, HLA-A1 antigens might well act as receptors for CPZ (Smeraldi an

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The possible role of prostaglandin E1 deficiency in the immunological abnormalities seen in schizoph‘self’-antigens in disordered autoimmune states. There are two sub-groups of T lymphocytes, the ‘helper’ cells which cooperate with B lymphocytes in the production of a strong and specific antibody response to foreign antigens, and the ‘suppressor’ cells. The suppressor cells control overactive B ly

預(yù)定

https://doi.org/10.1007/978-3-322-96467-0In the literature, very early reports on the deviations of serum proteins in schizophrenics can be found; for reviews see Fessel (1962) and Solomon and Moos (1964). However, there is very little information so far about how the observed changes are connected with schizophrenic disease, although different opinions have been presented.