Titlebook: Behavioral Neurogenetics; John F. Cryan,Andreas Reif Book 2012 Springer-Verlag Berlin Heidelberg 2012 behavioral disorders.behavioral gene

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John F. Cryan,Andreas ReifImportant volume on neurobehavioral genetics.Written by leading experts in the field.With numerous illustrations.Includes supplementary material:

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Current Topics in Behavioral Neuroscienceshttp://image.papertrans.cn/b/image/182868.jpg

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Market Trend Classification Using ML and DL, Due to the accumulated genetic knowledge the zebrafish has now been considered an excellent research tool in other disciplines of biology too, including behavioral neuroscience and behavior genetics. Given the complexity of the vertebrate brain in general and the large number of human brain disorde

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Shannon W. Anderson,S. Mark Young that few genetic and environmental factors act alone, but that instead they regularly act in concert to determine predisposition to psychiatric disorders. Quite a few cases now have been reported in which stratification of subjects by exposure to environmental pathogens has been shown to alter the

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Shannon W. Anderson,S. Mark YoungHT receptor, 5-HT transporter, synthetic and metabolic enzymes has been linked particularly to impulsive aggression. The current summary focuses mostly on recent findings from pharmacological and genetic studies. The pharmacological treatments and genetic manipulations or polymorphisms of a specific

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Addressing Mental Health Problems at Workr the twenty-first century. The present chapter will summarize existing rodent models for research in psychiatry, mimicking depression- and anxiety-related diseases. In particular we will highlight the use of selective breeding of rodents for extremes in stress-related behavior. We will summarize ma

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Promoting Mentally Healthy Workplaces of stress have shown parallel behavioral changes such as impaired working memory, cognitive flexibility and fear extinction. This coincides with morphological changes to pyramidal neurons in the prefrontal cortex, hippocampus and amygdala, key cortical regions mediating these behaviors. Increasing

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https://doi.org/10.1007/978-3-030-23455-3increase risk for developing the disorder. A consensus view is that the genetic component involves several common risk alleles of small effect and/or rare but penetrant copy number variations. Furthermore, there is increasing evidence for broader, overlapping genetic-phenotypic relationships in psyc

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