Titlebook: Basis and Treatment of Cardiac Arrhythmias; Robert E. Kass,Colleen E. Clancy Book 2006 Springer-Verlag Berlin Heidelberg 2006 cardiac arrh

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Experimental Therapy of Genetic Arrhythmias: Disease-Specific Pharmacology,gorithms. Finally, the most ambitious step in the study of genetic disease is to discover a novel pharmacological therapy targeted at correcting the inborn defect (locus-specific therapy) or even to “cure” the DNA abnormality by replacing the defective gene with gene therapy. At present, this curati

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https://doi.org/10.1007/978-3-030-39379-3r nodal re-entry and atrioventricular re-entrant tachycardia in hearts with an accessory atrioventricular connection. The components of the electrocardiogram, and of extracellular electrograms directly recorded from the heart, could only be well understood by comparing such registrations with record

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Yirakpoa Ikaba,Charles Enyindahficacy, but very frequently to minimize the likelihood of proarrhythmia. . avoiding proarrhythmia has become a key element of contemporary new antiarrhythmic drug development. Further, recognition of the magnitude of the problem has led to important advances in understanding basic mechanisms.While t

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Robert Ebo Hinson,Emmanuel Mogajidrugs. Very selective agents have great potential as tools for further study of the role the Na/Ca exchanger plays in arrhythmogenesis. For therapy, they may have their specific indications, but they carry the risk of increasing Ca. load of the cell. Agentswith a broader action that includes Ca. cha