Titlebook: Bacterial Protein Toxins; Klaus Aktories,Ingo Just Book 2000 Springer-Verlag Berlin Heidelberg 2000 Antigen.Eiweiss.Gift.bacterial protein

Capture

, Exoenzyme S, a Bifunctional Cytotoxin Secreted by a Type-III Pathway,c pathogen. (. et al. 1983; . et al. 1992; . et al. 1994). Although . can be controlled with antibiotics, the prevalence of multi-drug-resistant strains has limited the number of antimicrobial agents that are of clinical use (. 1972). . possesses an array of virulence factors that contribute to its

finale

HERTZ

Molecular Biology of Actin-ADP-Ribosylating Toxins,butes to many cell functions, such as movement, cytokinesis, endocytosis, exocytosis and control of intercellular junctions. Its disorganization by toxins leads to important cell dysfunctions and induces the severe lesions which are found in the associated pathologies. Some toxins modify enzymatic r

Conduit

濃縮

Molecular Biology of Large Clostridial Toxins,argest of the clostridial toxins are those produced by ., and .. These toxins, which include toxin A [molecular weight (M.) = 308 000 Da] and toxin B (M. = 270 000 Da) of ., toxin HT (hemorrhagic toxic; M. = 300 000 Da) and toxin LT (lethal toxin; M. = 260000 Da) of ., and .-toxin (M. = 250 000 Da)

DEVIL

The Cytotoxic Necrotizing Factor 1 from ,,acquisition of genes coding for virulence factors. Apart from those strains acting in the intestine, there are distinct . strains that have the ability to cause a wide variety of extra-intestinal diseases, including septicemia and meningitis in neonates and urinary infections in children and adults.

Accommodation

Shiga Toxins of , and , ,of diarrheagenic . called STEC (for Shiga toxin-producing .). Shiga toxins of STEC were formerly called Shiga-like toxins and are alternatively named Vero toxins (. et al. 1996; . et al. 1996). We favor the Shiga-toxin nomenclature system based largely on historical precedence. Indeed, the prototype

Stress-Fracture

Clostridial Neurotoxins, toxins A, B, C., D, E, F and G. Although the main effect of all of these toxins is a dysfunction of striated muscles, the symptoms of tetanus and botulism are diametrically opposed. Tetanus is characterized by recurring spasticity of antagonistic skeletal muscles, and it is these dramatic symptoms

MIRTH

粘土