Titlebook: Bacterial Invasiveness; Virginia L. Miller Book 1996 The Editor(s) (if applicable) and The Author(s), under exclusive license to Springer-

Commission

Parley

ORE

filial

Book 1996the molecular and genetic level, and the important signal transduction events are being elucidated. It is now becoming clear that there are both similarities and differences both in terms of the steps involved and of the genetic basis of bacterial invasiveness. These reviews of the "state of the art" provide a foundation from which to proceed.

悲觀

0070-217X both similarities and differences both in terms of the steps involved and of the genetic basis of bacterial invasiveness. These reviews of the "state of the art" provide a foundation from which to proceed.978-3-642-85218-3978-3-642-85216-9Series ISSN 0070-217X Series E-ISSN 2196-9965

最初

我正派

Uptake of Enteropathogenic , by Mammalian Cells,ases as well as mesenteric lymphadenitis, with outbreaks occurring primarily in cold-weather climates (. et al. 1991; . et al. 1990). The most notable characteristic of . infections is that mild or inapparent bacterial infections may trigger a number of autoimmune disorders (Saario et al. 1992; Toiv

接觸

Invasion and the Pathogenesis of , Infections,nd a strong inflammatory reaction indicate sites of infection (. et al. 1964). Clinical signs of shigellosis range from mild diarrhea to severe dysentery with blood, mucus, and pus in the stool. Epidemiological studies indicate that . are transmitted by the fecal-oral route and sometimes by contamin

對(duì)手

Molecular and Cellular Bases of , Entry into Host Cells,me . serotypes are highly adapted to a specific host (e.g., S. . and . can infect only human beings and poultry, respectively) or preferentially infect one species (e.g., S. . and . preferentially infect swine and cattle, respectively). In contrast, other serotypes can infect a broad range of hosts

酷熱

Molecular and Genetic Determinants Involved in Invasion of Mammalian Cells by ,,te in nonprofessional phagocytic cells. The process of entry into nonphagocytic cells is often referred to as “parasite-directed endocytosis”or “induced phagocytosis”, since it involves the participation of both the pathogen and the host eukaryotic cell. In those cases where it has been carefully st