Titlebook: Axonal Regeneration in the Mammalian Central Nervous System; A Critique of Hypoth Dorothy Eve Oorschot,David Gareth Jones Book 1990 The Edi

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無(wú)動(dòng)于衷

https://doi.org/10.1007/978-1-4939-7665-2 of surviving cells. This is not evident, however, for the mammalian central nervous system (CNS). The proliferation of neurons occurs mainly during fetal development and ceases once, differentiation is attained (Clemente 1964), so that new neurons cannot be formed following injury. Hence, axonal re

嚴(yán)峻考驗(yàn)

https://doi.org/10.1007/978-1-0716-3633-6 regenerative events in mammalian and submammalian vertebrates, as well as the comparison of mammalian developmental and regenerative events. The regenerative response of mammals has also been assessed using a variety of morphological and biochemical techniques; these studies primarily describe even

deadlock

Harsh H. Oza,Esther Ng,Daniele M. Gilkesl functions of microenvironmental components requires the development of animal model systems which allow the analysis and manipulation of individual components (Varon et al. 1983). In vitro systems often permit one to do this, since they permit greater control of the chemical and physical environme

objection

https://doi.org/10.1007/978-1-0716-3633-6nse. With respect to the hypotheses which seem more relevant to the limited regenerative response in the mammalian CNS (see Sect. 2.7), results from in vitro studies support the in vivo work in that the somal response, the presence for specific growth factors, compatibility between neuronal and non-

bizarre

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未成熟

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不遵守

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BRAVE

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flamboyant

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