Titlebook: Autoimmunity; Andrzej Górski,Hubert Krotkiewski,Micha? Zimecki Book 2001 Springer Science+Business Media New York 2001 Arthritis.Diabetes.

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書目名稱Autoimmunity影響因子(影響力)




書目名稱Autoimmunity影響因子(影響力)學(xué)科排名




書目名稱Autoimmunity網(wǎng)絡(luò)公開度




書目名稱Autoimmunity網(wǎng)絡(luò)公開度學(xué)科排名




書目名稱Autoimmunity被引頻次




書目名稱Autoimmunity被引頻次學(xué)科排名




書目名稱Autoimmunity年度引用




書目名稱Autoimmunity年度引用學(xué)科排名




書目名稱Autoimmunity讀者反饋




書目名稱Autoimmunity讀者反饋學(xué)科排名

畫布

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Complement Regulatory Proteins and Autoimmunity,indings suggest that complement regulatory proteins have dual roles at inflammatory sites: enhancement of cellular resistance to complement attack and acceleration of theclearance of cells injurious to the organism due to complement-mediated mechanisms. To assist the former function, a therapeutic a

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Evidence that Fas and Fasl Contribute to the Pathogenesis of Experimental Autoimmune Encephalomyelirse can be acute or chronic relapsing, characterized by a rapid onset of hind limb weakness that commonly progresses to paralysis, followed by spontaneous remission starting 7–10 days after the initial appearance of symptoms. EAE can also be induced passively by the adoptive transfer of . activated

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Exposition

Is Lack of Peripheral Tolerance Induction a Cause for Diabetes in the Non-Obese Diabetic Mouse?,ing naive T cells, the dendritic cells are also involved in generating central and peripheral tolerance to self molecules. In this process DC2 cells appear to be more important for the development of peripheral tolerance than DC1 cells. Besides abnormalities in the development of bone marrow-derived

Connotation

反對(duì)

The Role of T Cells in Rheumatoid Arthritis,ed CD4.CD28. T cells is fundamentally changed and is shifted towards tissue-injurious capabilities. CD4.CD28. T cells are particularly important in patients with extra-articular manifestations of RA, where they may have a direct role in vascular injury. Understanding the mechanisms underlying the lo

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