Titlebook: Autoimmune Thyroiditis; Approaches Towards i W. A. Scherbaum,U. Bogner,G. F. Bottazzo Conference proceedings 1991 Springer-Verlag Berlin He

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Association of HLA Class I and Class II Genes and Adrenal Steroid 21-Hydroxylase Gene with Subacute mmunity, but also specific recognition by the T-cell receptor supplemented by helper factors is necessary for an immune response [2]. Since organ-specific autoimmune diseases are chronic diseases, it might he speculated that the helper factors (interleukins), which provide a positive feedback, play

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A Search for Linkage Between Phenylthiocarbamide Tasting, the Kell Blood Group Locus and Autoantibodn the blood group Kell and TPO autoantibody. Kell has been reported to be linked to the locus governing the ability to taste phenylthiocarbamide (PTC) [3] which in turn may be associated with certain forms of thyroid disease [4]. In order to explore further the genetic basis of autoimmune thyroid di

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https://doi.org/10.1007/978-3-663-13372-8 the insulin-producing beta cells of the islets of Langerhans [1,2]. The immunological pathogenesis of this disease was initially suggested by the presence of a mononuclear cell infiltration of the islets, “insulitis,” in patients who had died within a few days of the clinical onset of IDDM [3,4]. T

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https://doi.org/10.1007/978-3-642-49213-6ted to Graves’ disease. Both diseases can be observed clustered in families [1]. Furthermore, some patients progress from Graves’ disease to Hashimoto’s thyroiditis, illustrating that both diseases are part of one spectrum of thyroid autoimmunity. Antibodies to microsomal antigen (thyroid peroxidase

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