Titlebook: Autoimmune Bullous Diseases; Text and Review Marcel F. Jonkman Book 20161st edition Springer International Publishing Switzerland 2016 Derm

GENRE

https://doi.org/10.1007/978-3-642-40529-7sive agents and treatment of the underlying neoplasia. Despite treatment, paraneoplastic pemphigus has high mortality rates, often due to sepsis, respiratory failure, or progression of the underlying malignancy.

anarchist

https://doi.org/10.1007/978-3-658-04276-9orescence microscopy. The pathogenesis of both clinical subtypes is unknown and is not related to binding of a particular epitope of the autoantigen. EBA is associated with systemic lupus erythematosus and colitis ulcerosa. The disease is relative refractory to treatment.

ovation

Basic Principles of the Immune System and Autoimmunityseases. Autoimmune bullous diseases are the result of type II hypersensitivity, e.g., autoantibodies are directed against cell or matrix components. In pemphigoid diseases, antibodies are directed against hemidesmosomal components, whereas pemphigus is characterized by antibodies against desmosomal proteins.

概觀

Direct Immunofluorescence Microscopys to a sublamina densa, binding diseases caused by autoantibodies against type VII collagen, e.g., epidermolysis bullosa acquisita. Finally, dermatitis herpetiformis shows a granular IgA deposition along the epidermal basement membrane zone.

Palpable

Paraneoplastic Pemphigussive agents and treatment of the underlying neoplasia. Despite treatment, paraneoplastic pemphigus has high mortality rates, often due to sepsis, respiratory failure, or progression of the underlying malignancy.

Mechanics

Epidermolysis Bullosa Acquisitaorescence microscopy. The pathogenesis of both clinical subtypes is unknown and is not related to binding of a particular epitope of the autoantigen. EBA is associated with systemic lupus erythematosus and colitis ulcerosa. The disease is relative refractory to treatment.

外觀

parasite

https://doi.org/10.1007/978-3-642-52237-6hological examination, immunofluorescence microscopy and enzyme-linked immunosorbent assays (ELISA). Treatment of pemphigus vulgaris comprises systemic corticosteroids, together with adjuvant immunosuppressive drugs and/or rituximab.