Titlebook: Atlas of Heart Failure; Cardiac Function and Wilson S. Colucci (Professor of Medicine, Chief) Book 2002Latest edition Springer Science+Busi

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Angiotensin-converting Enzyme Inhibition and Angiotensin Receptor Blockade,n activity provides an indicator for prognosis in heart failure patients [1]. Patients with mild and asymptomatic heart failure demonstrate relatively less activation, but even these values are increased compared with normal. The degree of renin activity is intensified in the presence of diuretic th

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,β-Blockers,y, as has been demonstrated with positive inotropic agents such as vesnarinone and flosequinan, chronic heart failure therapy may improve quality of life and reduce symptoms while increasing mortality. As discussed in Chapter 11, several large randomized controlled trials have demonstrated the benef

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New Approaches to the Treatment of Heart Failure,erwhelming evidence that angiotensinconverting enzyme (ACE) inhibitors and β-blockers achieve both of these goals and therefore are first-line therapy. For patients who remain symptomatic, the clinical utility of digitalis and spironolactone has also been established. Despite the appropriate use of

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Diastolic Dysfunction: Pathophysiology, Clinical Features, and Treatment,unction may also affect the right ventricle (RV), but there is relatively little information about RV diastolic dysfunction. Therefore this chapter focuses exclusively on LV diastolic dysfunction with the understanding that the same principles undoubtedly apply to the RV.

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Molecular and Cellular Events in Myocardial Hypertrophy and Failure,ers, and peptide growth factors. Genetic manipulations in small animal models are refining our understanding of the stimuli and molecular events that lead to progression of heart failure. Although much remains to be learned about how these stimuli interact with signaling pathways to regulate the rem

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Eleuterio Vallelado,Philip Molyneuxlume) pattern of ventricular growth. Although remodeling in response to a pathologic condition can in one sense be considered adaptive because it permits the restoration of pump function in the face of an imposed hyperfunctional condition, the extent of ventricular remodeling is nevertheless an impo