Titlebook: Atlas of Dermatology; Inflammatory, Infect Adriana Motta,Luis Fernando González,Mariam Rolon Book 2022 Springer Nature Switzerland AG 2022

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Inflammatory Diseases of the Blood Vessels with Cutaneous Compromiser destroys the vascular wall, causing bleeding and ischemia of the affected tissue, which can manifest as clinical findings in the skin [1]. These dermatological manifestations of vasculitis are diverse, and the etiological approach of each is carried out according to the caliber of the compromised blood vessel. Within this group are:

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Inflammatory Diseases Affecting Melanocytessenting as hypochromic or depigmented macules, and, conversely, those that activate melanin production within the melanocyte as a consequence of an inflammatory process that results in hyperpigmented macules. The pigmentary disorders of inflammatory origin are grouped as follows:

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Vesiculobullous Inflammatory Diseasesulcers. The pathophysiological mechanism of these entities involves an abnormal immune response to different components of the epidermis or dermoepidermal junction. The most representative diseases are thus grouped:

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From Logic Design to Logic ProgrammingIn this group of dermatological diseases are all entities that present clinically with papules or plaques due to erythema, desquamation, and lichenification. One of the representative entities of this group is psoriasis. There are different presentations of this same entity grouped as follows:

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Lecture Notes in Computer ScienceIn this group of dermatological diseases are entities that present clinically with papules or plaques with associated erythema, peeling, and lichenification. Within this group are lichen and lichenoid reactions grouped as follows:

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https://doi.org/10.1007/978-3-319-10434-8A number of skin diseases occur as a direct result of the activation of the immune system secondary to exposure to medications. Within this group are:

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An Invariance Principle for Parabolic SPDEsThis section documents inflammatory skin diseases that occur with erythema, erythematous-edematous wheals (urticaria), and purpura. The pathophysiological mechanisms often differ, despite sharing comparable clinical presentations with similar lesional distributions and morphology. Within this group are: