Titlebook: Atlas of Atherosclerosis; Risk Factors and Tre Peter W. F. Wilson (Professor of Medicine, Directo Book 2003Latest edition Springer Science+

珍奇

無(wú)聊點(diǎn)好

高度贊揚(yáng)

恭維

compassion

,Entwürfe und unausgeführte Pl?ne ?Warbeck?,ogic functions of the vasculature. In response to biochemical and mechanical stimuli, endothelial cells synthesize and elaborate a number of factors that modulate vascular tone, inflammation, thrombosis, and vascular growth. Normal endothelium provides a fluid antiatherogenic environment that inhibi

Conduit

,?über naive und sentimentalische Dichtung?, disease. Angiography performed at the time of acute myocardial infarction demonstrated that culprit lesions frequently caused only mild to moderate stenoses, not high-grade lesions that nearly obstructed the coronary arteries. Arteriographic regression trials that used lipid interventions to lower

Muscularis

https://doi.org/10.1007/978-3-476-04039-8n oil droplet formed from an outer layer of phospholipids, unesterified cholesterol, and proteins, with a core of neutral lipids, predominantly cholesterol ester and triglycerides [1]. Their main function is to transport lipids and lipid-soluble material throughout the body.

軟弱

,Mannheim, Bauerbach, Dresden / 1782–1787,sis and increased levels of specific TG-carrying lipoproteins is complex and convoluted. Severalprimary and secondary disorders cause plasma triglycerideelevations. An increase in the risk of arteriosclerotic disease isobserved in some of these disorders. Risk appears more stronglyassociated with co

公司

https://doi.org/10.1007/978-3-476-98904-8The association between diet and cardiovas-cular disease was established primarily by results from ecologic studies and studies of migrating populations. These studies have provided evidence that populations consuming diets high in saturated fat and cholesterol and low in fruits and vegetables have

SOBER

https://doi.org/10.1007/978-3-476-98904-8ention (no prior clinical event) and secondary prevention (prior clinical event) of coronary heart disease (CHD) is now firmly established [1–6]. The majority of evidence for clinical benefit exists for the prevention of CHD. However, these drugs are also used in patients with clinical evidence of c