Titlebook: Atherosclerosis III; Proceedings of the T G. Schettler,A. Weizel Conference proceedings 1974 Springer-Verlag Berlin Heidelberg 1974 atheros

GILD

Identification of Lipoproteins Involved in Human Atherosclerosis by conventional lipid ‘stains’ (such as oil red 0) and that of specific fluorescence due to LDL or VLDL at all stages of the development of atherosclerotic arterial lesions. But no similar relationship could be shown for HDL (Walton and Williamson, 1968). In this previous work: a) comparisons were

廢除

Quantitative Interrelationships between Plasma Constituents and Normal and Atherosclerotic Human Int. The cholesterol esters which accumulate in extracellular locations also appear to be derived directly from plasma, but those which accumulate inside fat-filled cells are probably formed by esterification . (Smith et al., 1967; Smith and Slater, 1972a). Using the technique of immunofluorescent micr

Intentional

Lipid Metabolic Pool in Subcellular Fractions of Rabbit and Human Atherosclerotic Lesionsestablished (Day and Wahlqvist, 1968, Wahlqvist et al. 1969). The intracellular lipid is present as lipid droplets, which differ in composition from the extracellular lipid (Smith et al., 1967). Two types of lipid droplets have been demonstrated in human atherosclerotic lesions, one isotropic, the o

congenial

Glucose and Lipid Metabolism in the Human Arteriosclerotic Aortaterial tissue and its changes in arteriosclerosis is limited. Though the ability of mammalian arterial tissue to synthesize fatty acids has been the subject of several reports (Whereat et al., 1967; Howard Jr., 1968; Filipovic and Buddecke, 1971; Filipovic et al., 1973), the lipid accumulation in hu

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高爾夫

Arterial Lipid Metabolism in Diabetic Animal Models with Reduced or Elevated Plasma Insulin Levelstic patients. The incidence of atherosclerotic disease is increased in hyperlipidemic diabetic populations but is relatively low with normal serum lipids (Rudnick and Anderson, 1962). Abnormal carbohydrate metabolism (Wahlberg, 1966) and hyperinsulinemia (Stout and Vallance-Owen, 1967) are common in

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,te that both the content and composition of arterial wall phospholipids and cholesteryl esters alter as a result of aging and of atherosclerosis. These observations have been supplemented by studies on the synthesis and metabolism of phospholipids and cholesteryl esters and it is now well-recognized