Titlebook: Asthma Treatment; A Multidisciplinary D. Olivieri,P. J. Barnes,G. C. Folco Book 1992 Springer Science+Business Media New York 1992 Atmen.P

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Potential Role of Mast Cell Proteases in Asthma,arks of asthma. Because known mast cell mediators may not explain adequately the various manifestations of asthma, the search continues! I will present evidence that the mast cell enzymes, tryptase and chymase, have potent effects on appropriate airway cells, suggesting that they may play key roles in the pathophysiology of asthma.

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https://doi.org/10.1007/978-3-658-10088-9thesize histamine and other chemical mediators implicated in the pathophysiology of a variety of inflammatory and immunological disorders [Marone, 1985; Marone .,1988; 1989a]. Both cell types also express plasma membrane receptors (FcεRI) that bind the Fc portion of IgE antibody with high affinity [Rossi ., 1990].

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,Kinematik des starren K?rpers,nd . (Manning et al. 1990; Holroyde et al. 1981; Griffin et al. 1983; Weiss et al. 1982; Barnes et al. 1984). Increased production of leukotrienes has been demonstrated following antigen challenge of the airways of allergic patients . (Creticos et al. 1984; Ishihara et al. 1985; Isono et al. 1985) and . (Dalhén et al. 1983).

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Neural Control of Airways,pathophysiology of asthma. Indeed there is a complex interaction between inflammation and neural control of airways. Autonomic nerves regulate many aspects of airway function and influence the tone of airway smooth muscle, airway secretions, blood flow, microvascular permeability and the migration a

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